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Abstract

Ivabradine is a specific funny current inhibitor, which blocks Hyperpolarization-activated Cyclic Nucleotide-gated cation channels intra-cellularly and results in delayed diastolic depolarization in a use-dependent manner. Prior-believed to be exclusively expressed within the sinoatrial node, funny channels were recently revealed to be also expressed in the atrioventricular node and throughout the myocardium. These invite a key clinical question as whether pharmacological effects of ivabradine may extend beyond its conventional clinical use.

Here reported is an unusual case of atrioventricular dissociation associated with ivabradine use, dissecting which may lead us to consider broader potential implications of this observation

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